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Where Is The Link Between Gout And Diabetes?

Gout is more common in people with type 2 diabetes than in those without. Gout is a form of arthritis that causes sudden swelling, pain, and stiffness in the joints. Gout can cause severe pain. It is usually felt in the big toe, which can turn very red. Gout attacks can also occur in the ankle, knees, and other joints. Gout attacks can last up to 10 days before subsiding. Gout attacks can last for as long as 10 days before the pain subsides. Nearly 85% of people who have experienced gout attacks once experience another within three years.


It is a buildup of uric acids in your bloodstream. Your body produces uric acid when it breaks down purines. Purines are a type protein found in many foods as well as all cells of your body. Uric acid is normally found in your blood. It passes through your kidneys, and is excreted when your urine is empty. If your blood level of uric acids is too high for a long time, it can form needle-like crystals which get stuck in your joints and soft tissues. Gout is characterized by painful, swollen joints.

Gout can be inherited from one generation to another, possibly because people in the same family have similar lifestyles. In a study published in The American Journal of Medicine (2010), thousands of adults were examined along with their children. Researchers found that people with higher levels of uric acid were more likely to develop type 2 diabetes. 71% more likely than women who have it to develop diabetes than those without it. Each condition can adversely affect the other. Other medical conditions can also cause elevated uric acid, including gout.

Good To Know

Gout patients, for example, have higher cholesterol, elevated triglycerides and high blood glucose levels. Gout can increase the likelihood of people developing type 2 diabetes, metabolic syndrome, or kidney disease. 90% of people with type 2 diabetes are overweight. It is not surprising that approximately 50% of those with gout are overweight. Being overweight (or carrying too much weight around your middle) can increase the levels of certain inflammatory substances. Gout attacks can be aggravated by this.

High blood pressure is a common problem in type 2 diabetes. High BP can lead to high levels of uric acid, which can lead to gout. Insulin resistance is another problem that high BP can cause, which is the bane of type 2 diabetics. Gout and diabetes are also linked to kidney disease and heart disease. Gout and type 2 diabetes are linked in a certain way. You can beat both with the same lifestyle and diet strategies that you use to beat diabetes.


How is gout diagnosed and treated? Your doctor will conduct a physical exam of the affected joints. Gout is diagnosed when the blood level of uric acid exceeds 415 umol/L (micromoles per litre). How can gout be treated? Resting the joint can help ease pain during an attack. You can also reduce pain by taking an NSAID (nonsteroidal-anti-inflammatory drug), such as naproxen or ibuprofen. Your doctor may give you a shot with a corticosteroid (such prednisone) to stop a gout attack. Orally, this can also be administered. Colchicine, an ancient medicine for gout, was first used in Ancient Egypt to treat rheumatism. It reduces the buildup of uric acid crystals, which causes pain in the affected joints. Colchicine is an alternative to NSAIDs for those who are unable to tolerate them. Side effects such as gastrointestinal upset can limit the use of colchicine at high doses.


What causes gout attacks? Every person is different, so what causes gout attacks in one person might not be the same for another. Gout can be made worse by drinking alcohol. If you already have gout, it can cause a sudden attack. Purines are a common ingredient in many beers. The liver produces uric acid when alcohol is consumed. Gout is a condition where alcohol stimulates the production of uric acid in the liver. You can reduce your risk of developing gout by losing weight. A balanced, healthy diet combined with regular exercise is the best way to safely lose weight. Drinking fluids can reduce the risk of crystal formation.

Eight large glasses of fluids per day is recommended (1.5 litres). You can count all drinks, other than alcohol, towards your fluid intake. This includes drinks that contain caffeine like tea and coffee. Caffeine can be mildly diuretic. It makes you urinate more often. Regular coffee and tea drinkers will develop a tolerance to caffeine, so they will experience mild dehydration if they consume more. Caffeine may not directly influence the risk of developing gout. Research has shown that people who drink coffee may have lower blood levels of the uric acid than those who drink the same amount of tea.


Drinking water should be a priority for diabetics. Research has shown that men who eat more vitamin C are less likely develop gout. Research also suggests that vitamin C can be taken as a dietary supplement (500-1000mg per day) to lower your blood uric acid levels. Vitamin C is a vitamin that helps your kidneys eliminate uric acid from the body. If you are considering adding vitamin C to your diet, and you are also taking prescription medicines, you should consult your doctor to ensure that the supplement is safe for you. Some people may experience loose stool if they consume high amounts of vitamin C.

The tart cherries, also known by their other name, are quite different from the sweet cherries. They are brightly red and retain their colour even after being juiced, frozen, or dried. Sweet cherries, on the other hand, are almost black in colour and can be either dark red or purple. Gout is treated naturally with sour cherries and sour grape juice. These cherries have been shown to reduce blood uric acid and inflammation associated with gout. Sour cherries can quickly deteriorate so it is important to freeze or dry them as soon as possible. The best way to enjoy them is dried, in smoothies, cereal bars, and baked goods. Your body produces uric acid by breaking down purines that you eat. Reduce your intake of purines-rich foods is a good strategy if you have gout.