摆脱痛风的最快方法是什么？这里有答案。
为了摆脱痛风，你必须降低尿酸水平。而降低高尿酸的最好方法是改善我们的饮食。消除提高尿酸的食物并引入其他对身体更健康的食物是非常重要的。首先要做的是避免富含嘌呤的食物。
降低尿酸的食物
为了降低高尿酸，我们必须始终保持身体的水分充足。每天喝1.5到2升的水，以刺激肾脏功能，因为提高尿酸的嘌呤会通过尿液排出。除了喝水之外，我们还可以饮用绿茶或蒲公英。可以吃的降低尿酸的肉类和鱼类包括鸡肉、火鸡、兔肉、鳎鱼、John Dory、鳕鱼和新鲜鳕鱼。换句话说，所有低脂肪的白色肉类和鱼类。
柑橘类水果，如橙子、柠檬和葡萄柚是降低高尿酸的理想选择。但其他水果，如樱桃、草莓、覆盆子、小红莓、苹果、葡萄和香蕉，也有利于降低尿酸水平。生的或烤的（但不是油炸的）坚果、乳制品和蔬菜，如芹菜、花椰菜、韭菜和青豆，在这方面也非常有益。
乳制品应该包括脱脂牛奶、不含奶油的低脂酸奶和不含脂肪的奶酪。此外，应包括橄榄油和种子油，如葵花籽油或玉米油，但不包括脂肪，如猪油、黄油或奶油。
有机种子对降低尿酸水平也非常好。亚麻、紫花苜蓿和大麦种子富含蛋白质、磷和镁，亚麻籽、奇亚或马尾草也是如此。如果我们有高尿酸的问题，所有这些都应该包括在我们的饮食中。
樱桃汁也是减少尿酸和预防痛风发作的一个很好的选择。
芹菜提取物已被发现是对抗与痛风和风湿病等疾病相关的疼痛的最有效的天然成分之一。具体来说，芹菜籽被发现有能力降低血液中的尿酸水平，以及提供强大的抗炎作用。要尝试这种家庭疗法，可从中医那里购买芹菜籽，将其放在水中煮至变软，过滤混合物并饮用一杯。
马尾草是一种具有药用价值的植物，作为一种伟大的天然清洁剂脱颖而出，因此它是治疗风湿病、关节炎和痛风等疾病的良好天然疗法。它有助于通过尿液排除多余的尿酸，并因其抗炎特性而缓解疼痛。准备输液，将一把干马尾草加入水中，煮沸后离火。待其冷却后，给自己倒一杯，开始注意其效果。
小苏打和水的制剂
降低高尿酸水平的一个很好的家庭疗法是准备小苏打和水的混合物。只要在半杯水中加入一小勺小苏打，就可以了。理想情况下，你应该用这种制剂每天喝八杯。然而，在服用的日子里，我们必须遵循低盐饮食，因为碳酸氢盐含有钠。
产生尿酸的食物
有几种食物会增加尿酸，或者如果我们患有这种问题就不应该吃。
内脏（肝脏、心脏和肾脏）、新鲜和罐装海鲜、油性鱼类（鲑鱼、金枪鱼或沙丁鱼）和红肉（牛肉、猪肉、牛肉或羊肉）是尿酸的禁忌食物。如果我们有太多的尿酸，我们就不应该在饮食中加入过多的尿酸。
这同样适用于豆类，无论是新鲜的还是干的，菠菜、绿叶蔬菜、芦笋、蘑菇、韭菜、香菇和酒精饮料，如啤酒。我们也不应该吃脂肪含量高的香肠，如腊肠或香肠。
对于高尿酸，我们必须控制食品的消费，如咖啡、工业糕点、脱水食品，如袋装汤和谷物以及由它们制成的产品。
总之，控制饮食是减少体内过量尿酸的一个关键因素。这是一个必须及时解决的问题，因为它可能导致严重的肾脏并发症并影响心血管健康。
按摩
在外部，另一个缓解痛风疼痛的家庭疗法是用薰衣草油按摩该部位。它的利尿和放松特性是减少痛风症状和疼痛的理想选择，当它们真的难以忍受时。它也是减轻肌肉紧张的理想选择。
适用于冷
对患处进行冷敷是缓解痛风疼痛的良好家庭疗法，但最重要的是，可以减轻炎症和肿胀。将冰袋或冷敷布直接放在关节处约15分钟。
什么是痛风？
Gout is a condition caused by an excess of uric acid in the blood, itself caused by the precipitation (= formation) of urate crystals in the cartilage. This accumulation causes severe joint inflammation. If left untreated, gout attacks escalate and eventually affect more and more joints.
What is the primary cause of gout?
Gout is caused by the improper breakdown of substances called purines. These are located in the nucleus of cells, then released when the cells themselves are destroyed. The purines are then transformed into uric acid, which can take the form of urate crystals. When a large number of cells break down, massive quantities of purines are released, uric acid levels soar and a gout attack is triggered. This cell fragility is typical of certain diseases, such as leukaemia or psoriasis, or their treatment, but can also be a side-effect of chemotherapy, for example.
That said, sometimes all it takes is a stressful event to cause a sudden increase in cell destruction. Uric acid can also increase in the body as a result of poor elimination, e.g. kidney failure. This is known as secondary gout. Primary gout is genetically programmed: it is caused by a faulty synthesis of certain enzymes. A large number of enzymes may be involved. One of them has been identified since it is known to be absent in children suffering from Lesch-Nyhan syndrome. These young patients suffer from gout attacks from an early age.
What factors influence blood uric values?
Gender: men suffer from hyperuricaemia 5 times more than women.
Age: uric acid levels in the blood increase with age. Hyperuricemia-related illnesses are therefore more likely to occur in men between 40 and 60 and in women in the menopause.
Weight: there is a causal relationship between gout and obesity. Dieting lowers blood uric acid levels, whereas fasting is unhealthy and leads to reduced uric acid excretion and a consequent increase in blood uric acid levels.
Diet: certain foods contain purines, the building blocks of uric acid, and should therefore be avoided: fatty foods, game, poultry, cold meats, shellfish and lobster, certain fish such as herring, anchovies, sardines and sprats, certain vegetables such as lentils, peas and beans, and especially meat from organs such as the liver, kidney and brain.
Alcohol: Alcohol abuse increases the level of uric acid in the blood and can even cause a gout attack. In patients suffering from gout, abstinence allows a huge drop in the uricemia value.
Medication: Certain medications, such as diuretics, can increase uricemia levels.
What conditions are associated with hyperuricaemia?
High levels of uric acid in the body are often due to obesity, high blood pressure or atherosclerosis. The harmful effects of uricaemia reinforce these risk factors. As a result, the risk of hyperlipidaemia, diabetes, high blood pressure and atherosclerosis increases considerably.
How is a gout attack treated?
Unlike other acute rheumatic conditions, gout is easy to treat. In the event of an attack, a non-steroidal anti-inflammatory drug is administered as the first line of treatment, which normally brings the attack to an end within five days. If this is not enough, it is not gout. Colchicine, a specific anti-inflammatory drug, is sometimes also prescribed.